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- Hang Yang1,4 na1,
- Yuan Jiao1,3 na1,
- Kefeng Wang1,4,
- Zhonghua Liu2,
- Peihong Wu1,5,
- Su Li1,3 &
- …
- Anxun Wang2
Scientific Reports volume 16, Article number: 25108 (2026) Cite this article
Correction to: Scientific Reports https://doi.org/10.1038/srep25918, published online 16 May 2016
This Article contains errors.
The representative fluorescence micrographs for the H2O2-treated SACC-83 cells in Figure 5A and Figure 5B were incorrectly selected during preparation of the final composite figure. Specifically, the representative H2O2-treated SACC-83 migration panel in Figure 5A was inadvertently duplicated from the SACC-LM migration panel in Figure 2A, and the representative H2O2-treated SACC-83 invasion panel in Figure 5B was inadvertently duplicated from the SACC-LM invasion panel in Figure 2B. The corrected Figure 5 is shown below.
SOD2-dependent production of H2O2 increases migration and invasion of SACC. To confirm that H2O2 production induces migration and invasion of SACC, SACC-83 cells were treated with H2O2 for 24 h. The migration and invasion of SACC-83 cells were significantly higher after treatment with 25 μM H2O2 once every 6 h (A,B). Adding H2O2 increased the protein concentrations of Vimentin, Slug, Snail, ERK, p-ERK, and MMP-2 and decreased E-cadherin protein concentrations and had no affection on expression of SOD2 (C).
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Chang, B., Yang, H., Jiao, Y. et al. Correction: SOD2 deregulation enhances migration, invasion and has poor prognosis in salivary adenoid cystic carcinoma. Sci Rep 16, 25108 (2026). https://doi.org/10.1038/s41598-026-65126-y
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DOI: https://doi.org/10.1038/s41598-026-65126-y